A common question people ask is “Is my weight determined by my genes?” This has led many to believe that there is no hope for them, and they are destined to be overweight forever. The truth of the matter is that our DNA does play a role in our weight, but it’s not as simple as just blaming your genes! In this article, we will discuss different ways you can change your genetic code so you can live the healthy life you deserve.
Why can some people eat anything they want and never put on any weight?
It’s not all directly connected to our genetic make up. Many people think that it is their dna which makes them gain weight. While there are certain genes linked to obesity, the majority of our DNA doesn’t directly affect us in this way.
Some people can eat whatever they want and never seem to gain weight. For others, it seems like every time they take a bite, they gain weight. What could be the reason for these differences?
Are Our Genes Responsible for Our Obesity?
In the USA the percentage of overweight and obese individuals has been steadily rising for over half a half a century. Is is not attributed solely to genetics though. The Centers for Disease Control and Prevention (CDC) highlights that genetic changes occur at a relatively slow pace for it to be considered an American obesity epidemic.
There are many other factors that are responsible for an increasing number of US citizens becoming overweight and obese.
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Sedentary lifestyles
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Processed foods
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Foods high in sugar
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Convenient and better transport
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Changes in working environments
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Emphasis on passive entertainment
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Easy diet fads
Researchers are still figuring out what combination of factors will lead to losing genetic fat, but it’s clear that the extent to which genes affect your outcome is complicated. Some people have more self-control than others and for them a simple diet isn’t going to cut it; they need specific plans tailored specifically towards their needs (and those don’t always include eating less). With this information in mind—as well as an understanding about how our environment impacts us too. You can make smarter choices when planning meals or exercising: knowing whether you’re at risk should help guide every decision!
The research team at Labmeeting have many articles on what causes weight gain and how to lose weight and what medication to use.
Obesity related genetic factors have also been found to be linked to DNA damage.
Many researchers agree on one thing though – if we want lasting results then ignoring any aspect or concentrating on just one wouldn’t be of value because even things like changing lifestyle habits have been shown to not reduce obesity levels that dramatically.
So, Genetic Obesity Links?
You may be genetically predisposed to being overweight if you have had a history of obesity and unsuccessful weight loss efforts. If both parents are overweight, there is an increased chance that this problem will carry on into your life as well; however, it can still be controlled with dieting or exercise consistently over time.
It is impossible to control all the factors that cause someone to be obese because there are so many genetic, psychological and environmental factors at play. Many people have been able to lose weight with a healthy diet and exercise plan but if you carry more body fat than others it may be due to your genetics.
What Genes are Responsible for Inherited Obesity?
The list of genes that are responsible for obesity is long and difficult to memorize. Some examples include:
PTPRG PTPRJ Amylin MCIR MCHR-R Leptin POMC GDNF NPY (neuropeptide Y) GHRL TRHR PCSKF KSR IGFBP-I GAD
FTO GENE
The FTO gene, a protein associated with fat mass and obesity is found on chromosome 16. Those who have this gene are more likely to become overweight because it leads them into adipose tissue accumulation which can lead someone else down an unhealthy path of becoming obese or even diabetic as well!
MC4R
A recent study that was conducted on human subjects has identified a gene known as “MC4R.” This particular gene controls how we use our food and contributes to feelings of hunger. It also impacts weight gain in those who are overweight due its ability regulate energy expenditure from different foods eaten throughout the day
ANKYRIN-B
Scientists have found a gene known for causing obesity in its variant form. When activated by this particular version, fat cells absorb glucose at an increased rate which causes them to grow larger than average sizes and pack on the pounds quickly!
PANX1
The presence of a genetic mutation that regulates obesity, called Panx 1 or pannexin1 can be correlated with higher risks for developing fatty liver disease. The outcome will vary based on what environmental factors are present in each person’s life; if they have high-sugar diets and don’t exercise then chances are greater than not there is something wrong!
IRX3
The Iroquois homeobox gene 3 (IRX3) is a gene that may cause difficulty losing weight. A study found individuals with deficient expressions of this one were 30% less likely to succeed at controlling their appetite, and can therefore experience higher rates for obesity problems such as type 2 diabetes or heart disease in later life due to inability retain fat loss despite trying hard on diets over many years
When IRX-type genes are not working properly due lack resources like food energy stores which promote body weight gain while up against lean tissue depreciation leading these lacking carriers live unhealthy lifestyles filled entirely by bad habits without any hope
Is it Possible to Alter These Genes?
Unfortunately, not all of these genes affect your weight; some are responsible for other problems like diabetes and heart disease. Even though you may be genetically inclined to obesity it doesn’t mean that nothing can be done about it . It is possible to change the genetic makeup by altering how they express themselves (called epigenetics).
For example, if someone is susceptible to obesity due to the FTO gene, research suggests that this risk factor can be mitigated with increased physical activity.
Who Suffers Most From Genetic Obesity?
There are a variety of factors to take into consideration when it comes to your genetic makeup causing you problems. Children, adolescents and young adults have the highest rates for obesity related disorders because they haven’t had enough time or education on how to live safely; they experiment with dangerous habits like smoking cigarettes without knowing that this simple act can become life-threatening.
Are Men More Likely Than Women to be Predisposed to Genetic Obesity?
Men are more likely to be affected by genetic obesity than women. This is due in part because men have higher levels of testosterone which can lead them into poor lifestyle choices that cause an increased risk for heart disease and type-two diabetes even if they never smoked a day in their lives!
What Can We Do To Prevent Genetic Obesity?
The best thing to do is remain informed about better lifestyle choices . For instance, if you are prone to obesity due your genetic makeup then it is best that you avoid high-fat foods and focus on healthy life decisions like eating whole grains instead.
Here are 3 simple measures you can take to prevent obesity
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Diet- diet is a critical element for preventing obesity and should include whole grains, vegetables and lean meats.Diet can also refer to eating healthy foods that promote weight loss through reducing fat intake from your diet. This type of diet may be used as part of a medical treatment plan in order to lose weight quickly or more effectively.
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Exercise – exercise is a great way to burn calories and lose weight. It can also help reduce risk factors for heart disease including high cholesterol, hypertension and diabetes. Exercise has been known to release endorphins which are chemicals that cause feelings of well being in our bodies!
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Healthy lifestyle choices – these are diet and exercise that can help to reduce obesity in susceptible individuals . This includes getting plenty of sleep, maintaining a healthy weight, staying hydrated with water, not smoking cigarettes or drinking too much alcohol.